Tylenol: not the safe drug we ever thought it was

I have been anti-Tylenol for almost 14 years.  I have seen what excessive use of Tylenol can do to the liver.  I have seen accidental over-ingestion that led to acute liver failure.  I have watched many die after intentional overuse of Tylenol products to end their life.  Acetaminophen, the active ingredient in Tylenol, is dangerous.  I have long said “if the FDA had to approve Tylenol today, they would surely say no and it would not be on the market”.

As you read through this article consider a few things.  Tylenol has been the “pain/fever reducer of choice” due to Reye’s syndrome risk in kids who took Aspirin, the drug of choice for PREGNANT women per their obstetrician, and it comes in liquid form to give babies/infants/children for illness and post vaccine fever/pain.  WHY was Tylenol the drug of choice?  I am starting to have some big concerns for why Tylenol was pushed so heavily.  Opiate medication is often mixed with Tylenol, such as hydrocodone and oxycodone.  Combination cold medications have Tylenol in them. Things such as Nyquil, Thera-Flu, Akla-Seltzer cold and flu……the list of drugs with Tylenol in them is EXTENSIVE.  So I have to consider a few things:  why did they want pregnant women taking this?  Why did they want kids/infants taking this?  Why is it mixed into so many multi symptom medications?  Why is it mixed into opiate pain medication?

We have discussed the dangers of vaccines and autism rates over the last few days, so this article is timely and should enter the discussion as well.  https://childrenshealthdefense.org/defender/tylenol-babies-children-vaccination-autism/

Here is a link to the paper questioning Acetaminophen and the neurological effects of it.  https://www.minervamedica.it/en/journals/minerva-pediatrics/article.php?cod=R15Y9999N00A22071303

“Our study shows that acetaminophen would never be approved for pediatric use by today’s regulatory standards,” Dr. William Parker, of WPLab, Inc., who led the research team that conducted the review on acetaminophen and autism, told The Defender.

The review, published in the July issue of Minerva Pediatrics, offers 17 lines of evidence that the commonly used remedy for pain and fever may be contributing to the autism epidemic.

The authors said their findings could have huge implications for preventing ASD, as acetaminophen is used so much in young children — in some populations, up to 90% of children receive acetaminophen in their early years.

The rate of ASD has skyrocketed in the last 40 years, and now affects 1 in 40 U.S. children.”

Parker and his colleagues previously conducted a systematic review, published in February in the European Journal of Pediatrics, which showed the studies claiming acetaminophen is safe for children did not examine the effect of the drug on neurodevelopment — even though the brain is one of the primary target organs for the drug’s therapeutic effect.

Their latest review builds on their earlier work showing acetaminophen was not assessed for impact on neurodevelopment, and on the strong evidence that prenatal exposure to acetaminophen increases the risks of neurodevelopmental issues.

OK full stop here:  pregnant women have, for YEARS, been told that the only pain reducing drug they can take “safely” during pregnancy is, you guessed it…..Tylenol.  And here is an article that discusses the dangers of taking Tylenol during pregnancy. https://www.nature.com/articles/s41574-021-00553-7

There is little research on the effect of postnatal exposure to acetaminophen.

However, according to the study, “circumstantial evidence regarding postnatal exposure to the drug is abundant, and includes at least three otherwise unexplained temporal relationships, data from laboratory animal studies, several miscellaneous and otherwise unexplained correlations, and a lack of alternative suspects that fit the evidence-derived profile.”

Based on this evidence, the authors “concluded without any reasonable doubt” that postnatal exposure to acetaminophen for children at risk is “responsible for many if not most cases of ASD.”

The key to understanding the role of acetaminophen in ASD is that acetaminophen alone does not trigger ASD — the negative impact happens in the presence of oxidative stress, according to Parker and his colleagues.

Oxidative stress is an imbalance of free radicals and antioxidants in the body that can lead to cell and tissue damage.

The body’s cells produce free radicals during metabolism, the chemical reactions that produce energy in our cells. These free radicals cause damage to cells and contribute to chronic illnesses and the aging process.

Unlike many drugs, some of the acetaminophen an individual takes is converted by the body into a highly toxic metabolite, N-acetyl-p-benzoquinone imine (NAPQI).

Usually, NAPQI is rapidly neutralized (rendered harmless) by glutathione, and because most healthy children have lots of glutathione, they aren’t harmed by NAPQI.

However, under conditions of oxidative stress, glutathione is depleted so the body can’t adequately remove the NAPQI. Left unremoved, the NAPQI reacts with a wide range of proteins, causing permanent damage to the proteins and associated cells.

Many babies and children are exposed to excessive oxidative stress through a variety of environmental and genetic factors including infection, vaccinatiions, treatment with antibiotics,  psychological stress, jaundice, heavy metals, problems with vitamin B metabolism, EMFexposure (wifi and cell phones), exposure to cigarette smoke or air pollution and exposure to organophosphates (pesticides, herbicides,etc). We vaccinate newborns and many these days develop jaundice; is it truly from the process of a newborns liver processing hemoglobin or is it from their fresh out of the womb vaccines??

How likely a child is to be injured by acetaminophen depends in part on the amount of oxidative stress present when the child takes the acetaminophen and the amount of the drug administered to the child.

According to the authors of the review, understanding the role of oxidative stress in combination with acetaminophen contributes to our understanding of the disparate risk factors for ASD: They are sources of oxidative stress.

So, if you loaded up a pregnant mom with Tylenol during pregnancy, we have already exposed a baby in-uterus to this drug with neurotoxic effects during key neuro developmental gestation periods.  Then, we add heavy metals and oxidative stress in the young infant/newborn when we vaccinate them and give them TYLENOL for pain/fever reducer post vaccine.  My lord, what have we done.